What are the symptoms of liver cirrhosis and ascites? The symptoms of liver cirrhosis and ascites are generally obvious. Ascites can also be said to be one of the main complications of liver cirrhosis diseases. It should be noted that because of liver cirrhosis, this disease cannot be completely cured. Therefore, it is critical to treat whether the liver sclerosis is timely and whether self -care during the treatment. It is recommended to learn more about the symptoms of liver cirrhosis. This is the key to whether it can be discovered in time. What you know is that this is one of the very common diseases in clinical practice.
What are the symptoms of liver cirrhosis and ascites?
1. Compensation period (generally belongs to Child-PUGHA level)
There can be a clinical manifestation of hepatitis and hidden onset. There may be mild fatigue, abdominal distension, mild swelling of the liver and spleen, mild jaundice, liver palm and spider mole.
2. During the period of breakdown (generally belongs to Child-PUGHB, C-Class)
Has liver damage and gate vein hypertension syndrome. (1) French symptoms, thinness, dull complexion, less urine, and edema of the lower limbs. (2) Symptoms of gastrointestinal symptoms, abdominal distension, gastrointestinal dysfunction, and even adverse syndrome, liver -sized diabetes, and symptoms such as urine and plenty of food can occur. (3) Bleeding tendency and anemia gums of gums, nose, purpura, anemia. Endocrine disorders, liver palms, skin pigmentation, feminine menstrual disorders, male breast development, and migrant glandular enlargement. Low protein hemophilia, two lower limbs, less urine, peritoneal effusion, and liver -sized thoracic effusion. Portal hypertension abdominal effusion, thoracic effusion, spleen large, hypertrophic spleen function, the establishment of the loop of the side veins, the esophageal-stomach veins, and the venous veins of the abdominal wall.
3. Check
Mild abnormalities in the compensation period, decreased serum protein during the loss period, rising globulin, and upside down in A/G. The original time of coagulant is prolonged, and the original activity of coagulant is reduced. Rising transaminase and bilirubin. Total cholesterol and cholesterol decreased, and blood ammonia can rise. The metabolism of amino acids is disordered, and the proportion of the branch/Fang is disordered. Elevated urea nitrogen and creatinine. Electrolytic disorders: low sodium, low potassium. Those who have recently occurred in the abdominal effusion and the rapid increase of the original abdominal effusion should do the cause of the abdominal cavity. The orthopedic effusion should be performed for routine tests, adenosine dehydaminase (ADA) measurement, bacterial culture, and cytology. In order to improve the positive rate of culture, the sampling operation of the abdominal effusion should be carried out by the bed, using a blood culture bottle, and the oxygen and anaerobic bacteria are used.
