Why is the esophagus gastric vein bleeding? Why?Why do you think it is safe?
1. Causes of bleeding from esophageal gastric venous veins
The liver has an important vein called the vein, which storage the blood of the intestinal vein, the subcontinal vein and the blood of the spleen vein. In addition, there are several side veins of the door vein, the most important of which is the gastric coronary vein (also known as the left vein of the gastric). The gastric coronary vein is connected with short veins of the gastric, esophageal veins, stagnation veins, semi -veins, and upper cavity veins. Under normal circumstances, the blood circulation of the door veins is smooth. The side branch veins of these door veins are in a closed state and passed without blood flow. However, if the liver (such as chronic hepatitis B, chronic hepatitis C, alcoholic hepatitis) due to some reasons (such as chronic hepatitis B, chronic hepatitis C, alcoholic hepatitis), the liver tissue structure is severely damaged, liver cirrhosis is formed, and the blood flow of the door venous Blood will generate back current, leading to the opening of the side branch veins above. Among them, the side branches of gastric coronary vein flow are open. Under the impact of strong vein pressure, there is a composite esophageal gastric vein. The surface of the esophageal gastrointestinal vein is covered with only one layer of mucous membrane. The shape under the gastroscopy is “earthworm -like”. The factors such as improper diet or abdominal pressure rises. Portal hypertension can also cause gastric mucosal ischemia erosion. This is collectively referred to as portal hypertensive gastric disease with the esophageal gastric vein song.
2. The manifestation of bleeding from esophageal gastric venous veins
The pathological basis of the hemorrhage of esophageal gastric vein is high pressure caused by liver cirrhosis. Common induction factors include eating rough food, severe nausea, vomiting, hiccups, cough, emotional excitement, sudden increase in abdominal pressure, taking some stimulating gastric mucous membranes that are irritating to gastric mucosa Drugs, etc., a small number of inducement can be followed. Those with mild bleeding or chronic bleeding can have no obvious symptoms, and sometimes only manifested as darkened stools. Those with severe bleeding can first have cold sweat, panic, dizziness, and pale complexion. After that, nausea occurs, vomiting coffee -like liquids, and even bright red blood. Some severe patients have shocks and endanger their lives. When patients with liver cirrhosis have inexplicable sweat, panic, pale face, etc., we must consider the possibility of bleeding from the esophagus gastric venous veins, and timely medical examination for treatment.
3. Prevention of bleeding from esophageal gastric venous veins
1. Actively treat the primary disease, remove the cause, prevent or slow the progress of liver hardening, and reduce the pressure of the gate. The most common cause of liver cirrhosis in my country is chronic hepatitis B and chronic hepatitis C, followed by long -term drinking. Patients with chronic hepatitis B and chronic hepatitis C should actively treat antiviral treatment under the guidance of a doctor to terminate or slow the development of the disease. Chronic hepatitis A antiviral can choose polyethylene glycol interferon and nucleoside drugs (such as Endonetcavir, Tonofoviri, and Delpidin), but linked liver hardening disable interferon; chronic hepatitis C antiviral can be based on virus genes according to the virus gene Choose different direct antiviral drugs (DAAS). Hepatitis C is currently a cure disease. Patients cannot take a good opportunity. Patients with alcoholic liver cirrhosis should taboo alcohol to avoid further damage to the liver and the direct stimulation of esophageal and gastric mucosa.
2. Patients with cirrhosis and varicose veins in the esophageal pipe should be mainly soft and rough foods, so as not to eat hard and rough foods, so as not to cause mucous membrane friction damage. The fritters, nuts, crispy fruits, etc. should be avoided as much as possible. Eating other foods should also pay attention to slowly. Fruit can choose soft fruits such as kiwi, bananas, watermelons, dragon fruit, etc., or crispy fruits such as apples and pears for fruit juice. Milk help protects the gastric mucosa. It can be consumed appropriately. Tea and coffee should not be too thick. The wine should be absolutely prohibited. Do not eat spicy foods to avoid stimulation of gastric mucosa.
3. Avoid rising abdominal pressure, because the increased abdominal pressure will increase the venous pressure of the door, which will cause the esophagus gastric venous veins to rupture and bleed. To this end, patients with cirrhosis are not suitable for participating in sports competitions and sports activities that require explosive power, such as throwing shot, throwing guns, weightlifting, high jump, long jump, etc. Things or bow down to move heavy objects; you cannot cough violently or frequently, if you have the above symptoms, you should try to relieve it in time. In addition, the stool should be kept unobstructed, and the stool is dry or constipation can easily cause the abdominal pressure to rise.
4. Careful medication. Patients who often reflect acid and qi can take acid suppression drugs such as omeprazole to reduce gastric acid corrosion of gastric mucosa. Careful use of blood circulation and stasis drugs and anticoagulant drugs, such as compound Danshen, saffron, aspirin, etc., those who need to use the drug should weigh the advantages and disadvantages, reduce or shorten the treatment. All kinds of analgesic drugs, such as ibuprofen, osoma, and acetaminol, have stimulating gastric mucosa, should be used as little or not as much as possible. If you need to take it after meals.
5. Surgical treatment if necessary. There are obvious hypertrophic spleen function (mainly manifested in the total number of white blood cells and platelets in blood routine), or those with gastrointestinal bleeding associated with door pulse hypertension should consider timely surgical or interventional surgery, because the foundation of liver cirrhosis is still there is still there is still there is still in the liver cirrhosis. The root cause has not been removed, so there will be a second and third time after a bleeding. The more the number of occurrences, the more difficult the bleeding is, the more dangerous. In this case, it is better to actively intervene.
Fourth, conclusion
